Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Wednesday, April 30, 2014

Plasma phospholipids identify antecedent memory impairment in older adults

This may be years down the road but I'd like to know so I can fasttrack my dementia prevention program. What does your doctor think and what is their dementia prevention program? Does your doctor remember your  33% chance of getting dementia/Alzheimers post stroke.

My program here:

1. Fish Oil Might Guard Against Loss of Brain Cells

2. Shakespeare and Wordsworth boost the brain, new research reveals

3. Physical activity is beneficial for late-life cognition

4. Omega 3 fatty acid for the prevention of cognitive decline and dementia

5. To keep sharp, retirees need to find a purpose

6.  Keep Working to Keep Dementia at Bay.

7. Preliminary medical testing results have shown that aspirin may prevent dementia and intestinal cancers

8.   THC(marijuana) may benefit those with Alzheimer’s disease.

9.  Common BP Drugs Tied to Lower Risk of Alzheimer's

10.  Study: Fruits and veggies may prevent dementia 

11.  Coffee May Lower Your Risk of Dementia

12. Light-to-moderate alcohol consumption, in comparison with abstinence, was associated with approximately 35-45% lower risk of cognitive decline or dementia.

13.  Activation of neuronal nicotinic acetylcholine receptors (nAChRs) has been shown to maintain cognitive function following aging or the development of dementia.

14.  Overeating is associated with an increased risk of mild cognitive impairment (MCI) in people 70 or older

15.  To Stave off Alzheimer’s, Stay Hungry?

16.  Calorie Restriction Prevents Neurodegeneration

17.  Researchers: Mini-Fast Prevents Alzheimer's

18. Evidence-Based Medicinal Properties of Coconut Oil - brain boosting



19.  Dementia Prevention – What Can Be Done? The generic medical version

     I bet your doctor gives you this.


I however am not going to wait for doctors and researchers to come up with a solution. You are going to have to read and decide for yourself how you want to approach dementia prevention. I doubt your doctor will be any help.


The possible test here:


http://www.nature.com/nm/journal/v20/n4/abs/nm.3466.html

Nature Medicine
20,
415–418
doi:10.1038/nm.3466
Received
Accepted
Published online




Alzheimer's disease causes a progressive dementia that currently affects over 35 million individuals worldwide and is expected to affect 115 million by 2050 (ref. 1). There are no cures or disease-modifying therapies, and this may be due to our inability to detect the disease before it has progressed to produce evident memory loss and functional decline. Biomarkers of preclinical disease will be critical to the development of disease-modifying or even preventative therapies2. Unfortunately, current biomarkers for early disease, including cerebrospinal fluid tau and amyloid-β levels3, structural and functional magnetic resonance imaging4 and the recent use of brain amyloid imaging5 or inflammaging6, are limited because they are either invasive, time-consuming or expensive. Blood-based biomarkers may be a more attractive option, but none can currently detect preclinical Alzheimer's disease with the required sensitivity and specificity7. Herein, we describe our lipidomic approach to detecting preclinical Alzheimer's disease in a group of cognitively normal older adults. We discovered and validated a set of ten lipids from peripheral blood that predicted phenoconversion to either amnestic mild cognitive impairment or Alzheimer's disease within a 2–3 year timeframe with over 90% accuracy. This biomarker panel, reflecting cell membrane integrity, may be sensitive to early neurodegeneration of preclinical Alzheimer's disease.

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