Changing stroke rehab and research worldwide now.Time is Brain! trillions and trillions of neurons that DIE each day because there are NO effective hyperacute therapies besides tPA(only 12% effective). I have 523 posts on hyperacute therapy, enough for researchers to spend decades proving them out. These are my personal ideas and blog on stroke rehabilitation and stroke research. Do not attempt any of these without checking with your medical provider. Unless you join me in agitating, when you need these therapies they won't be there.

What this blog is for:

My blog is not to help survivors recover, it is to have the 10 million yearly stroke survivors light fires underneath their doctors, stroke hospitals and stroke researchers to get stroke solved. 100% recovery. The stroke medical world is completely failing at that goal, they don't even have it as a goal. Shortly after getting out of the hospital and getting NO information on the process or protocols of stroke rehabilitation and recovery I started searching on the internet and found that no other survivor received useful information. This is an attempt to cover all stroke rehabilitation information that should be readily available to survivors so they can talk with informed knowledge to their medical staff. It lays out what needs to be done to get stroke survivors closer to 100% recovery. It's quite disgusting that this information is not available from every stroke association and doctors group.

Thursday, October 9, 2014

Coconut oil supplementation reduces blood pressure and oxidative stress in spontaneously hypertensive rats

I'm already using coconut oil as a dementia preventer. But never listen to me, your doctor knows everything even if you have to train them in their job.
http://www.biomedcentral.com/content/pdf/1753-6561-8-S4-P68.pdf
Background
Oxidative stress has been implicated in the pathogenesis
of hypertension and antioxidant compounds have been
used in the prevention and treatment of this disease. In
this context, coconut oil (CO) was found to have antioxidant
property due to its high polyphenol content. In
this study, we investigated the effects caused by chronic
treatment with CO on mean arterial pressure (MAP),
heart rate (HR), and lipid peroxidation in spontaneously
hypertensive rats (SHR).
Methods
SHR and their controls (Wistar Kyoto, WKY) were orally
treated with coconut oil (2 ml/day) or saline for thirty
days. Cardiovascular parameters were evaluated 24 hours
after the end of the thirtieth day. Serum oxidative stress
was measured by tiobarbituric acid reactive species assay
(TBARS).
Results and conclusions
The administration of CO was able to reduced the MAP
of SHR compared to SHR treated with saline (146 ± 11
n = 5 vs. 174 ± 6 n = 8 mmHg, respectively, p < 0.05).
On the other hand, treatment with CO had no effect on
MAP of WKY compared with WKY treated with saline
(136 ± 3 n = 6 vs. 113 ± 1 mmHg n = 8, respectively,
p < 0.05). In addition, CO supplementation reduced lipid
peroxidation in the SHR group compared to SHR + saline
(13.7 ± 2 vs. 27.5 ± 5 nmol/g n = 7, respectively, p < 0.05)
and also reduced in WKY compared with WKY treated
with saline (11.5 ± 1 vs. 21.9 ± 2 nmol/g n = 7 respectively,
p < 0.05). We concluded that treatment with coconut oil
(2 ml/day) for thirty days reduces the blood pressure and
oxidative stress in hypertensive rats.


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